Eric’s Hodgkin’s Lymphoma alternative treatment is a truly wonderful story of complete health restoration.
Eric R. was kind enough to share his Hodgkin’s Lymphoma alternative treatment triumph as a result of our comprehensive cancer care program at An Oasis of Healing. Watch the video below in its entirety to hear his story of how he was able to recover his health from the direst of circumstances!
We wanted to share a little of what Eric spoke about in his testimonial video below. We invite you to view the entire video as it is truly inspiring. It could help you or a loved one who may be dealing with cancer.
Eric and his wife Kristy travel across the US in an RV. Eric shared that he had been a healthy person up until his diagnosis of Hodgkin’s Lymphoma. He was having a constant cough that wouldn’t go away and decided to go to Urgent Care and check it out.
Urgent Care told him to get to an Emergency Center immediately. The reason for this was his blood count was extremely low at a 5.1. For men, an average blood count is 13. Following these tests, Eric was admitted to a hospital spending a week there.
The hospital ran tests and they showed that Eric had Hodgkin’s Lymphoma. He and his wife were shocked. Eric did not have the best experience while in the hospital. At that time they happened to be in Tucson, AZ, and Kristy while searching on the internet found An Oasis of Healing located in Mesa, AZ.
They got on the phone and made an appointment to meet Dr. Goodyear at An Oasis of Healing. Eric and Kristy meet some staff members and took a tour of our healing center. He said the center’s environment was amazing as soon as they walked in and completely different than the hospital and laying in bed all day.
Alternative Treatment For Hodgkin’s Lymphoma Helped Provide Eric R. With A Full Recovery As Told In This Video
Eric stated that he was treated like family at Oasis. He said the biggest blessing for him was when he met other patients going through the same types of challenges for their own situation. We all worked together having a common goal; helping and supporting one and other.
Eric suggests for those who are dealing with cancer or have a loved one dealing with it to come and visit An Oasis of Healing, meet Dr. Goodyear and staff and see firsthand for yourself why this place is like never you have ever seen before.
Dr. Nathan Goodyear, unfortunately, had to admit Eric to a hospital as a result of his kidney function. He had to spend six weeks there and another three weeks doing the rehabilitation. The hospital Eric was staying in told him that he would not get his kidney function back and it was probable that he wasn’t going to survive.
When the hospital released Eric, he met with Dr. Goodyear again and was afraid he would not take him back into the center for treatment of his Hodgkin’s Lymphoma as a result of his dire condition. Dr. Goodyear and An Oasis of Healing welcomed him back and immediately began a unique program developed specifically for him.
“Everyone at An Oasis of Healing is treated for their individual circumstances and I’m a great example of that”, said Eric. The program Dr. Goodyear had me on worked incredibly well and I got my kidney’s back! This enabled me to get off the dialysis machine sometime thereafter.
The doctor at the hospital treating Eric on the dialysis machine said that for people in his same condition receiving treatment for only three months would never come off it. Eric was not going to be one of them and got off dialysis from the Hodgkin’s Lymphoma alternative treatment and integrated therapies under the care of Dr. Goodyear and An Oasis of Healing.
Eric ended by saying, “He got his life back”. He was given his final Pet Scan and it showed no evidence of any disease! Seven months after his diagnosis in December of 2018 there was no longer any evidence of disease.
Eric R. recently shared how his treatment for Hodgkin s Lymphoma was a success while at An Oasis of Healing. Hearing his personal story of recovery was both emotional and inspiring! The tests came back that showed Eric he had Hodgkin s Lymphoma in December 2018. It was a shock to him and his wife. He was not having the best experience in the Hospital. As fate would have it, they were in Tucson, Az at the time and found An Oasis of Healing in Mesa, AZ. Eric said, you are treated like family here . He continued that the biggest blessing he received was meeting the other patients who were going through the same types of situations. Everyone works together with a common goal to help and support each other. Everyone here at this amazing center is treated individually and as a result of Dr. Goodyear s program, we got my kidney s back. I was able to get off of having dialysis after a certain number of weeks and had my port removed. Eric stated, he got his life back . He received his final Pet Scan and there is no evidence no disease! Eric was diagnosed in December of 2018 and seven months later there is no evidence of any disease. An Oasis of Healing 210 N Center St #102 Mesa, AZ 85201 480-834-5414 https://anoasisofhealing.business.site
When we get into the estrogen receptors and cancer, the complexity evident in hormones really increases.
The discussion of estrogen has to begin with the estrogen receptors because the estrogen receptors are critical in determining the signal that is transmitted from the different estrogens. Estrogen receptors and cancer is a complex subject that needs to be broken down and explained so a thorough understanding can be attained.
For the men in the audience, I can hear their collective: “I could have told you that…” But, for the women in the audience, at least it takes 2 because in men it only takes 1 hormone receptor to drive the show. The majority of the current research around estrogen receptors is in breast and prostate cancer. However, estrogen receptors are ubiquitous in the body and play a significant role in normal physiology [1].
The current understanding is that there are 2 estrogen receptors: estrogen receptor alpha (ER-alpha) and estrogen receptor beta (ER-beta). These two different estrogen receptor types appear to have some specific tissue-specific expression [2] and can produce profoundly different results when stimulated. As it relates to a wide variety of diseases, including cancer, ER-alpha has been shown to produce a pro-inflammatory [3] and pro-growth [4] signal from estrogen stimulation.
Inflammation is a prerequisite for cancer initiation and pro-growth signaling will favor cancer growth and progression.
After all, cancer is synonymous with unregulated growth. The often-mentioned ER+ in some cancer types is, in fact, a reference to ER-alpha. One cannot say all ER-alpha expression is associated with cancer, but the scientific literature does point to an increased cancer risk with increased ER-alpha expression and signaling.
ER-alpha expression and stimulation has been shown to play roles in breast cancer [5] and prostate cancer [6] to name a few. In fact, the loss of ER-beta expression and increase in ER-alpha expression has been shown to play a significant role in the initiation, growth, and invasion of prostate cancer [7], breast cancer [8], ovarian cancer and colorectal cancer [9]. Kind of important!
In contrast to ER-alpha, ER-beta has been shown to produce an anti-inflammatory [10] and anti-growth [11] signal from estrogen stimulation. ER-beta stimulation has even been shown to benefit asthma [12] and the excitotoxic diseases [13] MS, Parkinson’s disease, Alzheimer’s, and ALS. It doesn’t take a rocket scientist to see the potential positive impact on many diseases, including cancer, through ER-beta stimulated inflammation reduction and growth restriction to increase healthy and disease-free living potential.
Does ER-alpha always produce a pro-inflammatory signal? Does ER-beta always produce an anti-growth response? Are these receptors tissue specific? Can the expression of ER-alpha and ER-beta change? These are some of the questions that have yet to be answered by current science.
Similar to genetic expression and hormone expression, estrogen receptor expression can change. Maybe this can explain some of the variations in hormone response between individuals?
Or maybe this can explain the response difference and cancer risk in women to Hormone Replacement Therapy (HRT) in the years of pre-menopause, perimenopause and those that are post-menopause. The perfect example of the changes that can occur in ER expression and their effects on cancer risk can actually be found in men.
Estrogen receptors are an equal opportunity offender and in the case of men with low testosterone, there is a shift from ER-beta dominance to ER-alpha dominance in the prostate [14]. Remember, ER-alpha produces a pro-inflammatory and a pro-growth signal. In addition, inflammation up-regulates aromatase activity which increases testosterone to estrogen production to further increase estrogen production and stimulation of ER-alpha. What a vicious cycle!
The simple and linear thinking is that estrogen equals cancer in women and testosterone equals cancer in men.
No linear thinking here. The slogan is to keep it simple stupid. The problem is that ignorance of the complex for the purpose of the simple is more than stupid, it is dangerous. Accepting the complex as complex makes it simple. Moving on.
Keeping the focus on estrogen receptors in male cancer, the loss of ER-beta expression in the prostate has been shown to increase prostate cancer. In a recent study in mice, loss of ER-beta expression was found to increase abnormal prostate cell growth [15].
Another study in mice, found ER-beta stimulation lead to a decrease in prostate size and triggered programmed cell death through a process called apoptosis [16]. Apoptosis is a critical cancer control mechanism that is often lost early in cancer development.
However, we are talking about humans here. In human studies, ER-beta expression has been shown to reduce the incidence of prostate cancer [17][18]. Looks pretty promising that ER-beta expression in the male prostate is protective against prostate cancer and prostate disease.
Dr. Nathan Goodyear Explains The Complexity of Estrogen Receptors and Cancer in This Video
Just to stir things up a little more and to show the insane yet beautiful complexity that really exists in hormone receptors, I present Estrogen-Related Receptors (ERR). Estrogen-Related Receptors exist in 3 forms: alpha, beta, and gamma [19].
These receptors are nuclear receptors just like the regular estrogen receptors. These ERR have significant similarity to estrogen receptors however, they do not respond to estrogen as do estrogen receptors [20][21].
The exact specific substances that bind and stimulate the ERR are yet to be determined. This is a common theme in science: the body is beautifully and wonderfully made, yet we only know a fraction of it.
The implication of estrogen receptors and cancer is that it is not the hormone levels alone that poise the sole cancer risk for the individual as it is so often misunderstood and presented. It is the combination of the type of hormone present and its interaction with the type of estrogen receptors present plays a significant part in what signal the estrogen transmits: pro-inflammatory and pro-carcinogenic or not pro-inflammatory and not pro-carcinogenic.
[1] Tiwari-Woodruff S, Morales LB, Lee R, Voskuhl RR. Differential neuroprotective and anti-inflammatory effects of estrogen receptor (ER)alpha and ERbeta ligand treatment. Proc Natl Acad Sci U S A. Sep 11 2007;104(37):14813-8.
[2] Mueller SO, Korach KS. Estrogen receptors and endocrine diseases: lessons from estrogen receptor knockout mice. Curr Opin Pharmacol. 2001 Dec;1(6):613-9.
[3] Vegeto E, Cuzzocrea S, Crisafulli C, et al. Estrogen receptor-alpha as a drug target candidate for preventing lung inflammation. Endocrinology. 2009;151(1):174–184. doi:10.1210/en.2009-0876.
[4] Stephanie J Ellison-Zelski and Elaine T Alarid. Maximum growth and survival of estrogen receptor-alpha positive breast cancer cells requires the Sin3A transcriptional repressor. Molecular Cancer20109:263. Https://doi.org/10.1186/1476-4598-9-263.
[5] Ali S and Coombes RC. Estrogen receptor alpha in human breast cancer: occurrence and significance. J Mammary Gland Biol Neoplasia. 2000 Jul;5(3):271-81.
[6] William A. Ricke, Stephen J. McPherson, Joseph J. Bianco, Gerald R. Cunha, Yuzhuo Wang, and Gail P. Risbridger. Prostatic hormonal carcinogenesis is mediated by in situ estrogen production and estrogen receptor alpha signaling.The FASEB Journal 2008 22:5, 1512-1520.
[7] Weihua Z, Makela S, Andersson LC, et al. A role for estrogen receptor beta in the regulation of growth of the ventral prostate. Proc Natl Acad Sci U S A. 2001;98(11):6330–6335. doi:10.1073/pnas.111150898.
[8] Lazennec G, Bresson D, Lucas A, Chauveau C, Vignon F. ER beta inhibits proliferation and invasion of breast cancer cells. Endocrinology. 2001;142(9):4120–4130. doi:10.1210/endo.142.9.8395.
[9] Bardin A, Boulle N, Lazennec G, Vignon F, Pujol P. Loss of ERb expression as a common step in estrogen-dependent tumor progression. Endocrine-Related Cancer (2004) 11 537–551.
[10] Matthew C. Catley, Mark A. Birrell, Elizabeth L. Hardaker, Jorge de Alba, Stuart Farrow, Saleem Haj-Yahia and Maria G. Belvisi. Journal of Pharmacology and Experimental Therapeutics July 2008, 326 (1) 83-88; DOI: https://doi.org/10.1124/jpet.108.136275.
[11] Zhang J, Tu Y and Smith-Schneider S. Activation of p53, inhibition of telomerase activity and induction of estrogen receptor beta are associated with the anti-growth effects of combination of ovarian hormones and retinoids in immortalized human mammary epithelial cells. Cancer Cell Int. 2005 Mar 8;5(1):6.
[12] Matthew C. Catley, Mark A. Birrell, Elizabeth L. Hardaker, Jorge de Alba, Stuart Farrow, Saleem Haj-Yahia and Maria G. Belvisi. Estrogen Receptor β: Expression Profile and Possible Anti-Inflammatory Role in Disease. Journal of Pharmacology and Experimental Therapeutics July 2008, 326 (1) 83-88; DOI: https://doi.org/10.1124/jpet.108.136275.
[13] Tiwari-Woodruff S, Morales LB, Lee R, Voskuhl RR. Differential neuroprotective and antiinflammatory effects of estrogen receptor (ER)alpha and ERbeta ligand treatment. Proc Natl Acad Sci U S A. 2007;104(37):14813–14818. doi:10.1073/pnas.0703783104.
[14] Cohen PG. Obesity in men: the hypogonadal-estrogen receptor relationship and its effect on glucose homeostasis. Med Hypotheses. 2008;70(2):358-60.
[15] Imamov O et al. Estrogen receptor beta regulates epithelial cellular differentiation in the mouse ventral prostate. Proc Natl Acad Sci U S A. 2004;101:9375-9380.
[16] Krishnana G et al. Novel ER beta selective agonists induce prostate atrophy in rodents without affecting the hypothalamo-pituitary-gonadal axis. In the Endocrine Society Annual Meeting 2004. 2004. The Endocrine Society Press. Chase, Maryland, USA. 180-181.
[17] Leav I et al. Comparative studies of the estrogen receptors beta and alpha and the androgen receptor in normal human prostate glands, dysplasia, and in primary and metastatic carcinoma. Am J Pathol. 2001;159:79-92.
[18] Horvath LG et al. Frequent loss of estrogen receptor-beta expression in prostate cancer. Cancer Res. 2001;61:5331-5335.
[19] Coward P, Lee D, Hull MV, Lehmann JM. 4-Hydroxytamoxifen binds to and deactivates the estrogen-related receptor gamma. Proc Natl Acad Sci U S A. July 17 2001;98(15):8880-8884.
[20] Heard DJ, Norby PL, Holloway J, Vissing H. Human ERRgamma, a third member of the estrogen receptor-related receptor (ERR) subfamily of orphan nuclear receptors: tissue-specific isoforms are expressed during development and in the audit. Mol Endocrinol. Mar 2000;14(3): 382-92.
[21] Vanacker JM. Pettersson K, Gustafsson JA, Laudet V. Transcriptional targets shared by estrogen receptor-related receptors (ERRs) and estrogen receptor (ER) alpha, but not by ER beta. EMBO J. Aug 2 1999;18(15):4270-4279.
How the body processes hormones is called hormone metabolism.
Metabolism is presented as simply the means to gain or lose weight. Metabolism goes far beyond this simple definition. Metabolism is the mass production of day to day cellular processes that help the cell survive and thrive or not.
In reality, metabolism is a measure of optimal cellular and thus body homeostasis. Thus, metabolism can correctly be defined and applied to all cellular and body functions that result in day to day cellular homeostasis.
Hormone metabolites are just as important as the individual hormone levels and the hormone balance themselves. In fact, it may be more important as they reflect the flow, the trend of hormone movement which is so important in the assessment of function. We need to look at function, not static values. The body functions. The body is by no means static.
Hormone metabolites are just as they sound—the breakdown products that are the result of hormone metabolism. Whether hormones are endogenously made by the body or whether they are exogenously administered through hormone therapy, hormone metabolism manages the breakdown of these hormones to help the body survive, thrive or not.
The error is to think that hormone metabolites are merely waste products without any activity or physiologic significance. This has been the long-held thought. The truth is that hormone metabolites are very much active in hormone signaling.
In fact, hormone metabolites provide much of the risk and side effects equated to hormones and hormone therapy in cancer that is often overlooked. Not all hormone metabolites are bad. Some of these hormone metabolites can also provide significant protection.
Hormone Receptors
Hormone receptors are just what the words imply. They are the receptors that the individual hormones bind too. Once the hormones bind to the respective receptors, the signal of the hormone is then internalized inside the cell through a series of secondary messengers.
Hormone receptors are the doorway to the cell. Most of these signals then interact with DNA to turn genes off and/or on. There are, however, non-DNA and non-hormone receptor signaling that occurs as well.
Again, simple need not apply. As important as hormone receptors are to the signal transmission of hormones to the cell, they are impossible to assess except in the case of physical tissue specimens in biopsies.
Hormonal Regulation of Metabolism And Outside Influences
We are all products of our genetics (DNA) and our environment. The DNA that we have inherited from our parents is hard-wired. In contrast, the expression of DNA is not hard-wired but is in fact quite fluid.
The lifestyle choices we make and/or the environment we expose our DNA to influences the very expression of our DNA. Let this settle in for a moment. Though our DNA is fixed, we have the ability to determine whether genes are turned on or genes are turned off.
These effects can be good, or they can be bad. This is called the study of epigenetics or the study of things “above genetics”. Above genetics includes things i.e. diet, stress, sleep… that are above genetics, yet influence the expression of genetics. For example, studies have shown that our diet can affect our genetic expression to increase or decrease cancer risk [1]. Sorry, when your mom told you to eat your broccoli, she knew something that researchers are just now discovering. Broccoli anyone?
The same “environment” that can influence genetics can also influence the expression of hormones. One might call it the study of epihormones or “above hormones” if such a study actually existed. Based on the current dogma and outcomes of conventional medicine in hormones and cancer, it clearly should exist.
Physiologic Hormone Therapy When Treatment Is Required
The classic approach to most things these days is that more is better: super-size that drink, supersize those fries… Unfortunately, the same super-size logic applies to medicine today. This is no more apparent than in hormones. Just look at how conventional medicine handles low hormones: More is good, and a lot is even better.
The conventional approach to hormones is the same approach to chemotherapy: more is better; when the ole testosterone levels in men or estrogen levels in women are running on empty, you just stop off at your local doc station to filler up with more testosterone or estrogen.
The overdosing of these hormones is evident in the testing of these patients as well as the complications of these dosing strategies. If testosterone therapy causes cancer or thickens the blood (polycythemia) or causes aggressiveness/rage/anger issues, then why doesn’t endogenous testosterone production cause the same at age 24 when peak testosterone production occurs in men?
The point is that is doesn’t. These symptoms are simply the result of overdosing. If testosterone doesn’t cause these symptoms at the peak age of production of 24, then it shouldn’t with optimal physiologic dosing.
It only does with overdosing. The exact same can be said of estrogen and breast cancer. If testosterone caused prostate cancer and if estrogen caused breast cancer, then every young man and every young woman would have breast cancer.
They clearly don’t. The more is better philosophy, or the super-size logic need not apply to hormone therapy. That is if your goal is health and healing.
[1] Liu YC, Chen WL, Kung WH, Huang HD. Plant miRNAs found in the human circulating system provide evidence of cross-kingdom RNAi. BMC Genomics. 2017;18(Suppl 2):112. Published 2017 Mar 14. doi:10.1186/s12864-017-3502-3.
We are going to explore hormonal imbalance and cancer and why the body must be in homeostasis.
It only takes a pill. It just needs to be cut out. It is all about Estrogen. It is all about Testosterone. Disease is the normal outcome of life built into our genetic code without the means for change… Simple isn’t it.
When it comes to conventional medicine, when it comes to the conventional medicine marketing mantra, simple is the modus operandi. Simple would be nice. Simple, would be… well, simple. If only simple existed.
No matter how much we may want, the body just does not work on simple. The perfect example of this simple modus operandi can be found in the relationship between hormones and cancer.
Simple would have you believe men are nothing more than testosterone-fueled erections and women are simply estrogen-fueled hot flashes. Just look at the conventional medical marketing mantra: testosterone causes prostate cancer and estrogen causes breast cancer.
If a man is diagnosed with prostate cancer or a woman with breast cancer, testosterone or estrogen is the culprit and these hormones must be eliminated. It is almost a modern-day Wizard of Oz Lions and Tigers and Bears oh my… fear mongering.
Though our bodies may seem simple enough, they really are a beautiful, complex biochemical machine maintaining an intricate, delicate, balance designed for healing potential. Thank goodness for that complexity! Though advocated as simple, the body is anything but simple.
Dr. Nathan Goodyear Discusses Hormonal Imbalance And Cancer Goes In-Depth On The Important Subject Of Homeostasis
If it is not simple, then the opposite is true: complexity rules the day. Hormones, especially in reference to cancer, are a perfect example of this complexity. Hormones are just one piece of a very complex puzzle when it comes to hormones and cancer. For scientific, safe and effective hormone knowledge and therapy in patients with cancer it takes all the following:
Re-evaluate definition of normal
proper hormone evaluation
knowledge of hormone levels
hormone balance
hormone metabolites
hormone receptors
outside influences
physiologic hormone therapy
Re-evaluate Normal
The first question that often comes up in the discussion of hormones is are they normal. But what is normal after all? Who is normal? That discussion could go on for days.
Normal is defined as what falls within the statistical reference range of normal. Normal is then determined as a percentage of the population as a whole. This is why the definition of normal can change based on a shift in the population as a whole.
As the population becomes more abnormal, then a shift occurs as normal is re-defined. What was once abnormal is now normal and what is now normal is abnormal. Statistically speaking, the top 2.5% and the bottom 2.5% are determined to be statistically abnormal and all other 95% are deemed normal.
If I used that logic with my kids, a 65 on a test at school would be considered statistically normal right? From my perspective and my child’s, a 65 would not be considered normal. A 65 clearly falls far closer to failing than succeeding, yet it is statistically normal.
This highlights the limitations of this simple definition of normal when discussing anything, but especially hormone levels in people with cancer. This approach may work for disease medicine (though I highly doubt it), but it is not the best for the pursuit of healing from cancer.
Proper Hormone Testing And Evaluation
In the past, hormone evaluation was limited to blood. Today, hormone evaluations can come in all shapes and sizes: from blood to saliva, to urine. Even blood spot and urine spot hormone evaluation are available today.
People like my good friend, Dr. Zava, have pioneered these new hormone evaluation techniques. When it comes to different methods to evaluate hormones, no method is better than another, but it is all about perspective.
Each hormone evaluation is a different perspective or different window into the room of hormone physiology. No one window is better than the other, but each provides a different window with a different perspective.
The different windows provide different clinical perspectives that help to provide a more complete evaluation of hormone physiology of the individual. It also provides a more complete hormone treatment regimen for the specific environment of the individual with cancer.
The question simply becomes what perspective or what window provides the most useful information for the clinical question at hand. Is a single image good enough, or is a panoramic view better?
For me, I like the complete hormone panoramic view. In the treatment and healing of cancer, no questions should be left unanswered; or in this perspective, in viewed.
Hormone Levels
What is your number? The conventional medicine marketing mantra is full of this. Just look at the Testosterone marketing for men. What is your number is a common marketing cry for low Testosterone clinics around the country?
For this reason, I call testosterone clinics the 21st-century version of the methadone clinic. Contrary to most thought today, hormones are not just about the individual numbers.
Hormones are a means of communication throughout the body; a language if you will. I am always amazed at the answers I get when I ask post-menopausal women if they still make hormones or men older than 65 if they make testosterone.
Most will say no. Because of what they are exposed to in marketing, I am not surprised by their responses. Of course, the answer to both questions is yes.
The body must have these hormones to communicate, to live and to survive. Even after menopause, women need some estrogen.
It can be about the numbers if the numbers are exceedingly low or exceedingly high. However, that is a rare finding and usually occurs as a result of hormone therapy. Hormone impacts on individuals with cancer are rarely about the levels, but more about the balance.
Hormone Balance
Hormones must exist in balance. Balance is the source of health and wellness. Just look at the body for the importance of balance.
The human body is created in balance: 2 eyes, 2 ears, 2 legs…Just trying to lose a leg and see how the rest of your body is affected by the imbalance. Bumps, bruises, and even worse are definitely in the future.
The loss of balance is often the precipitating cause of symptoms short term and contribution to disease risk in the long-term. The most well-publicized hormone imbalance is that of the Estrogens and Progesterone in women. However, the imbalances of hormones are widespread in the body and have a significant impact on cancer risk and cancer healing.
Imbalances are not just isolated to hormones, but also apply to neurotransmitters and the immune system. The body must exist in balance.
